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Alsart (Valsartan)

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Alsart works by blocking the action of certain natural substances that tighten the blood vessels, allowing the blood to flow more smoothly and the heart to pump more efficiently. Alsart is in a class of medications called angiotensin II receptor antagonists.

Other names for this medication:
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Also known as: Valsartan


Alsart is an effective remedy against hypertension. Its target is to treat heart failure and to reduce the risk of death after a heart attack. It is working by preventing the hormone angiotensin II from narrowing the blood vessels, which tends to raise blood pressure. It is angiotensin II receptor antagonist. Alsart is also known as Valsartan, Valtan, Valzaar. Generic name of Alsart is Valsartan. Brand name of Alsart is Alsart.


To treat high blood pressure: 80 mg or 160 mg or more once a day. The maximum dosage is 320 mg a day. To treat heart failure: 40 mg twice a day. The maximum dosage is 320 mg daily. Take Alsart tablets orally with or without food. Do not crush or chew it. Take Alsart at the same time every day with water. If you want to achieve most effective results do not stop taking Alsart suddenly.


Seek emergency medical attention or call the Poison Help line at 1-800-222-1222.

Overdose symptoms may include fast heartbeats or fainting.


Store at room temperature between 15 and 30 degrees C (59 and 86 degrees F) away from moisture and heat. Keep your medicine container tightly closed. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Alsart are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


Hydrochlorothiazide may increase your risk of skin cancer. Avoid sunlight or tanning beds. Wear protective clothing and use sunscreen (SPF 30 or higher) when you are outdoors. Your doctor may want you to have skin examinations on a regular basis.

Do not use potassium supplements or salt substitutes, unless your doctor has told you to.

Drinking alcohol can further lower your blood pressure and may cause side effects.

Avoid driving or hazardous activity until you know how Alsart HCT will affect you. Your reactions could be impaired. Avoid getting up too fast from a sitting or lying position, or you may feel dizzy.

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Tumour necrosis factor-alpha regulates expression of the CCAAT-enhancer-binding proteins (C/EBPs) alpha and beta and determines the occupation of the C/EBP site in the promoter of the insulin-responsive glucose-transporter gene in 3T3-L1 adipocytes. We report the design, synthesis and characterization of a novel molecular beacon (MB-FB) which uses the fluorescent bases (FB) 2-aminopurine (AP) and pyrrolo-dC (P-dC) as fluorophores. Urolithiasis in rats with diabetes insipidus (Brattleboro strain rats). Hormonal regulation of cytokine release by human fetal membranes at term gestation: effects of oxytocin, hydrocortisone and progesterone on tumour necrosis factor-alpha and transforming growth factor-beta 1 output. We examined the expression of IL-21, IL-17, and interferon (IFN)-gamma in ITP patients and controls by enzyme-linked immunosorbent assay and flow cytometry. The anomalies in placental insertion and invasion, such as placenta previa and the various forms of abnormally invasive placenta, are today a rising obstetric pathology. We aimed to explore: (1) the inverse relationship, that is whether psoriasis might represent an independent predictor of OSA and its severity considering important predisposing factors and (2) the psoriatic phenotype related to severe OSA. This led us to test whether forced hepatic expression of SRC-1 can prevent the nonthyroidal illness syndrome in vivo. It was established that at this stage human choroid plexus primordium is an area of active morphogenesis. The majority of anthropometric parameters have shown significant age- and sex-specific differences in favour of male subjects, with exception of skin-folds that were apparently higher in female subjects.

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The acute transplanted rat leukemia L 5222 was used to investigate the two-drug combinations 1-(2-chloroethyl)-3-cyclohexyl-1-nitrosourea (CCNU) and cyclophosphamide (CPA) as well as methyl-CCNU and CPA. Cisplatin-induced macroautophagy occurs prior to apoptosis in proximal tubules in vivo. Electron microscopic studies showed the presence of increased number dark epithelial cells, expansion of intercellular area, loosening of the basal membrane and defects in it with invagination of the epithelial cells into the submucous layer. Moreover, we performed in vitro experiment to explore the cellular mechanism of VEGF protection effect. The PKR expression level was enhanced by IFN in all cells, but eIF2alpha phosphorylation level was not changed, probably due to the absence of double-stranded RNA species. Chemotherapy was administered consisting of three courses of an R-CHOP regimen, and 18F-FDG PET and CT scan revealed no signs of involvement 3 months after initiation of the chemotherapy. Bridging semantics and syntax with graph algorithms-state-of-the-art of extracting biomedical relations.

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